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Anti βngf Antibodies, supplied by R&D Systems, used in various techniques. Bioz Stars score: 92/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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recombinant human nerve growth factor  (Alomone Labs)
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human recombinant nerve growth factor  (PeproTech)
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Human Recombinant Nerve Growth Factor, supplied by PeproTech, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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human βngf  (R&D Systems)
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Podoplanin: a potential new player in the NGF/TrkA signaling pathway. (a) Real-time monitoring for potential human podoplanin and human <t>NGF</t> macromolecular interactions using SPR data obtained in a BiaCore apparatus showed a relatively high-affinity binding <t>of</t> <t>recombinant</t> NGF to immobilized podoplanin protein, as expressed in relative binding units on the y axis vs time. No binding was observed when a control GDF-8 recombinant protein was used even at concentrations five times higher than those used for the NGF assay. A heparin-based solution was used as a dissociation buffer. (b) No differences in the basal levels of total Ezrin or p-Ezrin (c) were observed between wild-type and podoplanin−/− mice hippocampal neurons. However, primary-cultured hippocampal neurons presented with comparatively lower levels of p-Ezrin upon NGF stimulation (50 ng/mL, 20 min) (d). Comparably, no differences in the levels of TrkA were observed under basal untreated conditions (e) but reduced levels were found in neurons from podoplanin−/− after NGF treatment (50 ng/mL, 2 h) (f). (g) Levels of CREB were also enhanced upon NGF stimulation in neurons from wild-type mice whereas CREB levels did not alter in response to NGF treatment in podoplanin−/− cultured neurons. Data are displayed as mean ± SEM. ns (p > 0.05), *(p ≤ 0.05), **(p ≤ 0.01), ***(p ≤ 0.001).
Human βngf, supplied by R&D Systems, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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recombinant human βngf rhβ ngf  (R&D Systems)
94
R&D Systems recombinant human βngf rhβ ngf
Podoplanin: a potential new player in the NGF/TrkA signaling pathway. (a) Real-time monitoring for potential human podoplanin and human <t>NGF</t> macromolecular interactions using SPR data obtained in a BiaCore apparatus showed a relatively high-affinity binding <t>of</t> <t>recombinant</t> NGF to immobilized podoplanin protein, as expressed in relative binding units on the y axis vs time. No binding was observed when a control GDF-8 recombinant protein was used even at concentrations five times higher than those used for the NGF assay. A heparin-based solution was used as a dissociation buffer. (b) No differences in the basal levels of total Ezrin or p-Ezrin (c) were observed between wild-type and podoplanin−/− mice hippocampal neurons. However, primary-cultured hippocampal neurons presented with comparatively lower levels of p-Ezrin upon NGF stimulation (50 ng/mL, 20 min) (d). Comparably, no differences in the levels of TrkA were observed under basal untreated conditions (e) but reduced levels were found in neurons from podoplanin−/− after NGF treatment (50 ng/mL, 2 h) (f). (g) Levels of CREB were also enhanced upon NGF stimulation in neurons from wild-type mice whereas CREB levels did not alter in response to NGF treatment in podoplanin−/− cultured neurons. Data are displayed as mean ± SEM. ns (p > 0.05), *(p ≤ 0.05), **(p ≤ 0.01), ***(p ≤ 0.001).
Recombinant Human βngf Rhβ Ngf, supplied by R&D Systems, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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anti human betangf  (R&D Systems)
90
R&D Systems anti human betangf
Podoplanin: a potential new player in the NGF/TrkA signaling pathway. (a) Real-time monitoring for potential human podoplanin and human <t>NGF</t> macromolecular interactions using SPR data obtained in a BiaCore apparatus showed a relatively high-affinity binding <t>of</t> <t>recombinant</t> NGF to immobilized podoplanin protein, as expressed in relative binding units on the y axis vs time. No binding was observed when a control GDF-8 recombinant protein was used even at concentrations five times higher than those used for the NGF assay. A heparin-based solution was used as a dissociation buffer. (b) No differences in the basal levels of total Ezrin or p-Ezrin (c) were observed between wild-type and podoplanin−/− mice hippocampal neurons. However, primary-cultured hippocampal neurons presented with comparatively lower levels of p-Ezrin upon NGF stimulation (50 ng/mL, 20 min) (d). Comparably, no differences in the levels of TrkA were observed under basal untreated conditions (e) but reduced levels were found in neurons from podoplanin−/− after NGF treatment (50 ng/mL, 2 h) (f). (g) Levels of CREB were also enhanced upon NGF stimulation in neurons from wild-type mice whereas CREB levels did not alter in response to NGF treatment in podoplanin−/− cultured neurons. Data are displayed as mean ± SEM. ns (p > 0.05), *(p ≤ 0.05), **(p ≤ 0.01), ***(p ≤ 0.001).
Anti Human Betangf, supplied by R&D Systems, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Podoplanin: a potential new player in the NGF/TrkA signaling pathway. (a) Real-time monitoring for potential human podoplanin and human NGF macromolecular interactions using SPR data obtained in a BiaCore apparatus showed a relatively high-affinity binding of recombinant NGF to immobilized podoplanin protein, as expressed in relative binding units on the y axis vs time. No binding was observed when a control GDF-8 recombinant protein was used even at concentrations five times higher than those used for the NGF assay. A heparin-based solution was used as a dissociation buffer. (b) No differences in the basal levels of total Ezrin or p-Ezrin (c) were observed between wild-type and podoplanin−/− mice hippocampal neurons. However, primary-cultured hippocampal neurons presented with comparatively lower levels of p-Ezrin upon NGF stimulation (50 ng/mL, 20 min) (d). Comparably, no differences in the levels of TrkA were observed under basal untreated conditions (e) but reduced levels were found in neurons from podoplanin−/− after NGF treatment (50 ng/mL, 2 h) (f). (g) Levels of CREB were also enhanced upon NGF stimulation in neurons from wild-type mice whereas CREB levels did not alter in response to NGF treatment in podoplanin−/− cultured neurons. Data are displayed as mean ± SEM. ns (p > 0.05), *(p ≤ 0.05), **(p ≤ 0.01), ***(p ≤ 0.001).

Journal: Annals of Medicine

Article Title: The brain-tumor related protein podoplanin regulates synaptic plasticity and hippocampus-dependent learning and memory

doi: 10.1080/07853890.2016.1219455

Figure Lengend Snippet: Podoplanin: a potential new player in the NGF/TrkA signaling pathway. (a) Real-time monitoring for potential human podoplanin and human NGF macromolecular interactions using SPR data obtained in a BiaCore apparatus showed a relatively high-affinity binding of recombinant NGF to immobilized podoplanin protein, as expressed in relative binding units on the y axis vs time. No binding was observed when a control GDF-8 recombinant protein was used even at concentrations five times higher than those used for the NGF assay. A heparin-based solution was used as a dissociation buffer. (b) No differences in the basal levels of total Ezrin or p-Ezrin (c) were observed between wild-type and podoplanin−/− mice hippocampal neurons. However, primary-cultured hippocampal neurons presented with comparatively lower levels of p-Ezrin upon NGF stimulation (50 ng/mL, 20 min) (d). Comparably, no differences in the levels of TrkA were observed under basal untreated conditions (e) but reduced levels were found in neurons from podoplanin−/− after NGF treatment (50 ng/mL, 2 h) (f). (g) Levels of CREB were also enhanced upon NGF stimulation in neurons from wild-type mice whereas CREB levels did not alter in response to NGF treatment in podoplanin−/− cultured neurons. Data are displayed as mean ± SEM. ns (p > 0.05), *(p ≤ 0.05), **(p ≤ 0.01), ***(p ≤ 0.001).

Article Snippet: Recombinant carrier-free human βNGF (R&D Systems, Minneapolis, MN, 256-GF-100/CF) was used to test the interaction of PDPN and NGF.

Techniques: Binding Assay, Recombinant, Control, Cell Culture